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Furthermore, we provide first data that the GSH level in human neurons may be mediated by NMDAR activation since the protective effect of enhanced network activity could be blocked with an NMDAR antagonist APV, consistent with the previous report (Fig

Among potential candidates, fingolimod, a sphingosine-1-phosphate receptor (S1P) modulator that prevents lymphocyte egress from the lymph nodes, has shown encouraging outcomes in preclinical and early clinical studies [340]

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